Ethical Issues in Journalism - 2922 Words

1.0 INTRODUCTION: Journalists are professional people, trying to work within a code of professional ethics. This includes the need to be fair to all parties involved in any news story. However, journalists cannot operate in a vacuum, doing what they think is right without pressures being put on them. Journalists face pressure from a variety of sources, all trying to make the journalist behave in a way which is not the way the journalist would choose. It is important that you try to resist all these forms of pressure, as far as possible. Of course, you will sometimes fail. This is an imperfect world, and journalists are also imperfect. Nevertheless, you should always try to resist the kinds of pressure. Some of the issues†¦show more content†¦What should you do about this kind of pressure? The first thing which any junior journalist should do is to report it to their editor. It will be for the editor to decide what to do. He or she will need to resist the employer as far as possible, pointing out the dangers of failing to report the news fully and fairly. The main danger is that readers or listeners, who already know of the event which is being suppressed, realize that it is not being reported and so lose confidence in the newspaper, radio or television station involved. This may, in the long run, result in more serious problems for ministers than some short-term embarrassment, and may do more long-term damage to your organization’s finances than the loss of one advertiser. The truth is, though, that your power to resist pressure from your employer is limited. You can only do your best, and accept that the rest is beyond your control. 2.3 Authority Both government-owned and commercial news media may face pressure from authority - the government, the police, customs, or some other branch of authority. Governments can threaten, or make, laws to force all news media to be licensed. This would give them power to grant licenses only to those news organizations which please the government. Even the threat to introduce such legislation may be enough to frighten journalists, andShow MoreRelatedThe Ethical Issues On Civil Journalism2970 Words   |  12 Pagescapabilities, there is an urgent need to identify news sources that are genuinely journalistic.’ (Meyers, Wyatt, Borden, Wasserman.) What I wish to bring out and elaborate in this paper are the ethical issues on how civil journalism, despite the acceptance and cooperation with professional journalism, is differentiated from its professional and vetted track. With the growth of technology and the ability to capture images, videos, and sounds along with the ease to distribute it to others eitherRead MoreAn Analysis of Quinns Accepting Manipulation or Manipulating Whats Acceptable?1578 Words   |  6 Pagesobjectivity of journalism with the coming of the digital age, and the increasing role that digital/information technology plays in media that reports or represents reality. Quinn begins his 2004 paper with the mention of the transition of non-digital/traditional photography into mainly digital photography with respect to journalism. More than just the changing of the photographic platform and format, Quinn notes the role that computer software that specializes in photographic manipulation in journalism. EarlyRead MoreEssay on The Watergate Scandal1199 Words   |  5 Pages June 17, 1972 forever changed both journalism and politics. A simple botched break-in marked the downfall of President Richard Nixon, and the rise to glory of two obscure young Washington Post journalists: Carl Bernstein and Bob Woodward. While their investigative journalism revealed the truth, their questionable methods and ethics have led to these questions; Do the ends justify the means? Was their behavior ethical and legal? The Watergate Scandal was a major political scandal duringRead MoreAndrew Stephens s Beyond News : The Future Of Journalism891 Words   |  4 Pagesobjective because we have biases and a variety of conflicts that we bring to our jobs. Mitchell Stephens, author of Beyond News: The Future of Journalism offers an argument against objectivity. Stephens states that objectivity is impossible because as much as one may try to disappear from the work, there is a kind of meditation that takes place in journalism no matter what, (117). By selecting who to interview or which side of a complex political dispute to air, journalists are evaluating and judgingRead MoreEssay on Codes of Practice1513 Words   |  7 Pagesworkforce we align with our ethical values to our employers, their values are shown through a company’s codes of practice. A code of practice is adopted by a profession or by a governmental or non-governmental organization to regulate that profession. A code of practice may be styled as a code of professional responsibility, which will discuss difficult issues, difficult decisions that will often need to be made, and provide a clear account of what behaviour is considered ethical or correct or rightRead More1) What Ethical Principles Should a Reporter Follow in Reporting/Writing a News Story to Ensure It Meets the Standards of Professional Journalism?1454 Words   |  6 Pagespeople are place importance on journalism ¡Ã‚ ¦s ethics. It is because everything that a journalist writes or says, or neglects to write or to say, in some or other way has an influence on many people, and that influences can be good or bad. The principles of good journalism are directed toward bringing the highest quality of news reporting to the public, this fulfilling the mission of timely distribution of information in servic e of the public interest. Therefore, ethical rhetoric is a important factorRead MoreEthical Dilemmas Regarding Technology : Ethical Dilemma1596 Words   |  7 Pages Ethical Dilemmas Regarding Technology An ethical dilemma is a situation where one must choose between two conflicting options, which are all morally correct. It is worth to note that ethics and morals are not easy to separate, as they both define what human behaviour should be at all times. The two aspects address issues of right and wrong as pertains to different walks of life. Cultural norms usually determine the ethical behaviour of a society, and these norms would inform people’s take on rightRead MoreAnalysis Of Ray Rice Domestic Violence1565 Words   |  7 Pagesthey reported on the incident fairly, they said that both Ray Rice and his wife made mistakes, and they did not favor the victim or the perpetrator. The last statement in the video could not be more true, it is not about them it is about the issue. The issue of domestic violence should be talked about repeatedly anytime a case like this happens. I feel that if you commit an act of domestic violence you should pay for it. It does not matter to me if you are a celebrity, athlete, politician, or JoeRead MoreCritical Analysis Of Burned And Shattered Glass1730 Words   |  7 Pagesespecially in regard to specific disciplines. (n.d.) In the field of journalism and communications, there is no difference. Ethics helps practitioners in this field fulfill the purposes expected of the media. These purposes include but are not limited to enlightening the public and safeguarding the rights of the individual. Professionals in the media have a responsibility to report the truth and express accountability. An axiom of journalism is to seek truth and report it. However, many individuals struggleRead MoreThe Code Of Ethics Of The Society Of Professional Journalists1054 Words   |  5 Pagesaccountable. Each of them considers a list of normative guidelines to orient the ethical decision-making process within a newsroom. In any case, the SPS’ orientations are framed by a utilitarian approach to ethics: Due to the voluntary character of t he code, the Code attempts to advising more than requiring and, in that sense, the recommended procedures and activities in which journalists should engage to accomplish an ethical work are oriented to journalists as individuals instead of a collective. In

Discovering the Occupations of Your Ancestors

Do you know what your ancestors did for a living? Researching ancestral jobs and occupations can teach you a great deal about the people who make up your family tree, and what life was like for them. An individuals occupation may give insight into their social status or to their place of origin. Occupations can also be used to distinguish between two individuals of the same name, often an essential requirement in genealogy research. Certain skilled occupations or trades may have been passed down from father to son, providing indirect evidence of a family relationship. Its even possible that your surname derives from the occupation of a distant ancestor. Finding An Ancestors Occupation When researching your family tree, it is usually fairly easy to discover what your ancestors did for a living, as work has often been something used to define the individual. As such, occupation is an often listed entry in birth, marriage and death records, as well as census records, voter lists, tax records, obituaries and many other types of records. Sources for information on your ancestors occupations include: Census Records - A good first stop for information on your ancestors job history, census records in many countries—including the U.S. census, British census, Canadian census, and even French census—list the primary occupation of at least the head of household. Since censuses are usually taken every 5-10 years, depending upon the location, they may also reveal changes in working status over time. If youre U.S. ancestor was a farmer, the U.S. agricultural census schedules will tell you what crops he grew, what livestock and tools he owned, and what his farm  produced.   City Directories - If your ancestors lived in an urban location or larger community, city directories are a possible source for occupational information. Copies of many older  city directories can be found online on subscription-based  websites such as Ancestry.com and  Fold3.com. Some free sources of digitized historical books such as  Internet Archive also may have copies online. Those that cant be found online may be  available on microfilm or through libraries in the area of interest. Tombstone, Obituary and other Death Records  - Since many people define themselves by what they do for a living, obituaries generally mention the individuals former occupation and, sometimes, where they worked. Obituaries may also indicate membership in occupational or fraternal organizations. Tombstone inscriptions, while more brief, may also include clues to occupation or fraternal memberships.   Social Security Administration - SS-5 Application RecordsIn the United States, the Social Security Administration keeps track of employers and employment status, and this information can generally be found in the SS-5 application form that your ancestor filled out when applying for a Social Security Number. This is a good source for the employers name and address of a deceased ancestor. U.S. Military Draft RecordsAll males in the United States  between the ages of 18  and 45  were required by law to register for the World War One draft throughout 1917 and 1918, making WWI draft records a rich source of  information on millions of American males born between about 1872 and 1900, including occupation and employment information. Occupation and employer can also be found in World War II draft registration records, completed by millions of men living in America between 1940 and 1943. Wills and  probate records, military pension records, such as Civil War union pension records, and death certificates are other good sources for occupational information.   What is an Aurifaber? Occupation Terminology Once you find a record of your ancestors occupation, you may be puzzled by the terminology used to describe it. Headswoman and hewer, for instance, are not occupations you commonly come across today. When you run across an unfamiliar term, look it up in the Glossary of Old Occupations Trades. Keep in mind, that some terms may be associated with more than one occupation, depending upon the country. Oh, and in case you are wondering, an aurifaber is an old term for goldsmith.   What Made My Ancestor Choose This Occupation? Now that youve determined what your ancestor did for a living, learning more about that occupation may provide you with additional insight into your ancestors life. Begin by trying to determine what might have influenced your ancestors choice of occupation. Historical events and immigration often shaped the occupational choices of our ancestors. My great-grandfather, along with many other unskilled European immigrants looking to leave behind a life of poverty with no promise of upward mobility, immigrated to western Pennsylvania from Poland in the early 20th century, and found employment in the steel mills and, later, the coal mines.   What Was Work Like for My Ancestors? Finally, to learn more about your ancestors day-to-day work life, you have a variety of resources available to you: Search the Web by occupation name and location. You may find other genealogists or historians who have created engaging Web pages full of facts, pictures, stories and other information on that particular occupation. Old newspapers may include stories, ads, and other information of interest. If your ancestor was a teacher you may find descriptions of the school or reports from the school board. If your ancestor was a coal miner, you may find descriptions of the mining town, pictures of the mines and miners, etc. Thousands of different historical newspapers  from around the world can be accessed online. Fairs, festivals, and museums often afford the opportunity to watch history in action through historical reenactments. Watch a lady churn butter, a blacksmith shoe a horse, or a soldier recreate a military skirmish. Take a tour of a coal mine or a ride a historic railroad and experience the life of your ancestor first hand. How to Learn Your Ancestors Occupation Visit your ancestors hometown. Especially in cases where a lot of residents of a town held the same job (a coal mining town, for example), a visit to the town can offer the chance to interview older residents and learn some great stories about day-to-day life. Follow up with the local historical or genealogical society for even more information, and look for local museums and displays. I learned much about what life was probably like for my great-grandfather through a visit to the Frank Sylvia Pasquerilla Heritage Discover Center in Johnstown, PA, which re-creates what life was like for the Eastern European immigrants who settled the area between 1880 and 1914. Look for professional membership societies, unions, or other trade organizations related to your ancestors occupation. Current members can be a great source of historical information, and they may also maintain records on the occupation, and even past members.

Mghf Free Essays

string(77) " showed a change from the baseline in the maximal chorea score of the UHDRS\." Huntington’s disease is an autosomal dominant (Harper et al, 1991) neurodegenerative disorder characterized by involuntary movements, cognitive loss, and psychiatric problems as described by Martin and Gusella (1986). These symptoms are related to the death of medium spiny projection neurons in the caudate nucleus, putamen, and the cortex (Reiner et al, 1988). In later stages of the disease, even areas such as the hippocampus and hypothalamus gets affected as well (Kassubek et al, 2004). We will write a custom essay sample on Mghf or any similar topic only for you Order Now Huntington’s disease has a prevalence rate of approximately 1 in 10,000 Caucasian populations (Harper et al, 1991). Patients with Huntington’s disease most typically display a choreic movement disorder involving involuntary writhing movements observed by George Huntington himself. The age of onset of Huntington’s disease seem to be normally distributed around the average age of 35 to 42, with small number of cases developing in patients younger than 20 years of age or older than 60 years of age (Andrew et al, 1993). The majority of juvenile patients, whose disease onset are at age 20 years or less, have inherited the paternally defected gene (Andrew et al, 1993). Patients usually died within 15 to 20 years of disease onset. Gusella et al (1983) first determined the location of the genetic mutation in Huntington’s disease to be the short arm of chromosome 4. It was after another 10 years did the researchers of The Huntington’s Disease Collaborative Research Group (1993) able to discover that a gene in chromosome 4 containing a trinucleotide repeat of CAG was abnormally expanded in diseased individuals. The IT15 (interesting transcript 15) gene, dubbed the huntingtin gene by the group, codes for the huntingtin gene. This CAG repeat, which translates into a poly-glutamine stretch, is highly polymorphic among the population ranging from 11 to 34 copies on normal individuals. However, in individuals with Huntington’s disease it expanded to more than 42 repeats and increasing to upwards of 100 (The Huntington’s Disease Collaborative Research Group, 1993). This provides evidence showing that the mutant huntingtin protein seems to be toxic to its native cells and confers a disease state to individuals with an extended length of repeats. There is also a correlation between the CAG length and disease onset as shown by Andrew et al (1993). The mutated elongated huntingtin protein is cut by enzymes into fragments and the fragments begin to form abnormal clusters, neuronal intranuclear inclusions (NIIs), inside cells. These clusters can also act to recruit normal proteins to adhere together as well (Davies et al, 1997). This was originally thought to cause the pathogenesis of Huntington’s disease. However, more recent studies have shown that the presence of NIIs is actually a coping response to the toxicity of mutant huntingtin proteins and acts to prolong the life of the cells and reduce intracellular mutant huntingtin in neighbouring neurons (Arrasate et al, 2004). The exact function of the wild-type huntingtin protein are unclear, however many efforts have been made in understanding its native functions. Nasir et al (1995) showed that homozygous huntingtin homologs in mice died before embryogenesis could occur and that heterozygotes displayed similar deficits as diseased human patients. Wild-type huntingtin is also crucial for establishing and maintaining neuronal identity, especially in cortex and striatum (Reiner et al, 2001). Current data can provide the conclusion that normal huntingtin protein has actions important for development in mammals. In vitro, wild-type huntingtin have been shown to act to protect brain cells from apoptotic stimuli, such as serum deprivation, mitochondrial toxins, or the transfection of death genes (Cattaneo et al, 2005). Wild-type huntingtin protein, not mutated, stimulates brain-derived neurotrophic factor (BDNF) production by acting at level of Bdnf transcription. BDNF is very important for survival of striatal neurons (Cattaneo et al, 2005). Intracellularly, huntingtin protein has been found to associate with various organelles such as the nucleus, endoplasmic reticulum, and Golgi complex (Cattaneo et al, 2005). It has also been found in neurites and at synapses, where it associates with vesicular structures and microtubules (Li et al, 2003). This characteristic has been shown to enhance vesicular transport of BDNF along microtubules (Gauthier et al, 2004). On a similar note, huntingtin interacts with a number of cytoskeletal and synaptic vesicle proteins that are essential for exo- and endocytosis at synaptic terminals. Wild-type huntingtin binds directly to the Src homology 3 domain of postsynaptic density protein 95, which binds NMDA and kainite receptors. This activity is decreased in mutant proteins and can lead to overactivation or sensitization of NMDA receptors (Cattaneo et al, 2005). Aside from the toxicity of the mutated huntingtin protein, the loss of normal huntingtin protein also seems to add to the pathogenesis of Huntington’s disease. Presence of only mutant huntingtin protein results in massive apoptotic cell death in the testes of male mice (Leavitt, 2001). However, no apoptosis can be seen in testes of mice expressing human mutant huntingtin when wild-type huntingtin is expressed as well (Leavitt, 2001). It was also seen that in mice, the absence of wild-type huntingtin protein led to a worsening of striatal atrophy and neuronal loss, and a significant decrease in neuronal cross-sectional area compared to mice that had wild-type huntingtin present (Cattaneo et al, 2005). Huntington’s disease still remains incurable to this day. However, many treatments are available for treatments of its symptoms. Chorea, the hallmark of the disease is a major target for many treatments. Such drugs include dopamine-depleting agents, dopamine antagonists, benzodiazepines, glutamate antagonists, acetylcholinesterase inhibitors, dopamine agonists, antiseizure medications, cannabinoids, lithium, deep brain stimulation and fetal cell transplantation (Frank and Jankovic, 2010). One notable drug currently in use is Tetrabenazine, which is the only US FDA-approved drug for treatment of Huntington’s disease (Frank and Jankovic, 2010). The drug acts by reversibly inhibiting the central vesicular monoamine transporter type 2, this cause a depletion of dopamine (Bagchi, 1983). The main area of effect for tetrabenazine is in the caudate nucleus, putamen, and the nucleus accumbens, all areas known to be responsible for the major pathology of the disease. The Huntington Study Group (2006) was able to demonstrate the efficacy of tetrabenazine in a double-blind, placebo-controlled trial. Subjects who received tetrabenazine showed a change from the baseline in the maximal chorea score of the UHDRS. You read "Mghf" in category "Essay examples" Compared to the baseline, treatment resulted in a reduction of 5. 0 units in chorea compared with a 1. 5 unit reduction for the placebo group. A second symptom targeted for treatment to allow patients to function normally is the psychiatric problems. Dopamine receptor blocking agents are commonly used as anti-psychotics in order to treat psychosis associated with Huntington’s disease (Frank and Jankovic, 2010). There are many ethical issues that have been associated with Huntington’s disease patients, in particular pertaining to the application of genetic testing. The expanded CAG repeats associated with Huntington’s disease (Gusella et al, 1983) are used as a predictive testing to determine the risk of a person for developing inherited HD gene. The debate of ethics with respect to genetic testing for Huntington’s disease seems to lie on several major factors including: autonomy, beneficence, confidentiality, and justice (Huggins et al, 1990). The question remains whether or not the current use of genetic predictive testing is ethical. Autonomy refers to the respect for the individual’s right to make an informed decision about an action that may have a profound effect on his or her life. The patients should feel no pressure from physicians or institutions with regards to their decision for genetic testing. Also, they should also have a full understanding of the consequences and implications of their decisions. This would require physicians to provide all the necessary information to the patients so that they are informed about the testing. If a woman refuses to get tests done for her baby even though her relatives are with her insisting on getting it performed, the physician cannot allow the testing to be performed. Beneficience is summarized by the phrase â€Å"first do no harm† (Huggins et al, 1990). This is an important factor when the results of the test may reveal genetic information about oneself. This implies not only avoiding harm to patient but also preventing harm to other individuals, which may include family members of patients. Due to the genetic characteristics of Huntington’s disease, family members can be both directly and indirection influenced by the results of the test. If for example a pregnant woman decides to get an ultra sound for her baby in order to find out if the baby has spina bifida, and upon confirmation by the physician decides that she wants to give birth at home as opposed to in a hospital where the baby could be treated correctly in order to minimize the risk of the disease. The physician should take any action he can in order to avoid any risk to the health of the baby. Confidentiality with genetic tests applies the same as with any other form of personal information. It should not be disclosed to any other third parties, which includes family members as well. For example, if two siblings were to go and receive a CAG repeat test at the same time and one sibling asks the physician about the results of the other sibling. The physician cannot disclose any information or they will violate the confidentiality of the patient. Justice simply means equal access to health services and information to all. This also includes long-term support and guidelines for testing as a medical service so that the service is not difficult to obtain if needed. For example, even though Cystic Fibrosis is mostly only prominent in Caucasians, the testing should still be offered to regnant women of other ethnicities as well. Other ethical dilemmas still exist with current genetic testing technologies. One particularly pertaining to Huntington’s disease is whether or not the information is obtained even though there is no cure for the disease. In diseases such as phenylketonuria (PKU), after newborn screening, dietary interventions will allow the individuals wit h the condition to lead healthy and normal lives (Lea et al, 2005). However, for Huntington’s disease, even after knowing that the disease will affect the individual what can be done? Therefore, would it be better to not know at all? By testing and finding out that an individual has the mutation that will eventually lead to the development of Huntington’s disease, the individual can attend to the initial development of symptoms and as a result may be treated much earlier. This can result in a more favourable prognosis. Similarly, the individual can live more cautiously as to prevent further exacerbation of their condition. On a more subjective level, the individual may use the knowledge to live a more fulfilling life knowing that he or she may not live as long as the rest of the population. Conversely, the information can also be used negatively. There can be a lot of discrimination in terms of applying for employment and health insurance. This information would also impact the ambition and life goals of the individual, the amount of psychological harm that comes with eventually developing an incurable and lethal neurodegenerative disease is extremely large. Similarly, would they still pursue to have a family and children if they knew that they had a very high risk of dying at a young age? This particular issue should be resolved by following the ethical factors listed before. As long as the individual is informed, are being prevented from harm, information not shared with any third party members, and has access to healthcare services, then being tested for Huntington’s disease will be ethically correct. This knowledge will give them insight on how to live the rest of their life. Another dilemma that has Harper, P. S. , Morris, M. J. , Quarrell, O. , Shaw, D. J. , Tyler, A. , and Youngman, S. (1991). The epidemiology of Huntington’s disease. J. Med. Genet. 89: 365-376. Martin, J. B. , and Gusella, J. K. (1986). Huntington’s disease: pathogenesis and management. N. Engl. J. Med. 315: 1267-1276. Andrew, S. E. , Goldberg, Y. P. , Kremer, B. , Telenius, H. , Theilmann, J. , Adamn, S. , Starr, E. , Squitieri, F. , Lin, B. , Kalchman, M. A. , Graham, R. K. , and Hayden, M. R. (1993). The relationship between trinucleotide (CAG) repeat length and clinical features of Huntington’s disease. Nat Genet. 4: 398-403. The Huntington’s Disease Collaborative Research Group (1993). A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington’s disease chromosomes. Cell. 72: 971-983. Gusella, J. F. , Wexler, N. S. , Coneally, P. M. , Naylor, S. L. , Anderson, M. A. , Tanzi, R. E. , Watkins, P. C. , Ottina, K. , Wallace, M. R. , Sakaguchi, Y. A. , et al. (1983). A polymorphic DNA marker genetically linked to Huntington’s disease. Nature. 306: 234-238. Reiner, A. , Albin, R. L. , Anderson, K. D. , D’Amato, C. J. , Penney, J. B. , and Young, A. B. (1988). Differential loss of striatal projection neurons in Huntington disease. Proc. Natl Acad. Sci. USA. 85: 5733–5737. Kassubek, J. , Gaus, W. , and Landwehrmeyer, G. B. (2004). Evidence for more widespread cerebral pathology in early HD: and MRI-based morphometric analysis. Neurology. 62: 523-524. Nasir, J. , Floresco, S. B. , O’Kusky, J. R. , Diewert, V. M. , Richman, J. M. , Zeisler, J. , Borowski, A. , Marth, J. D. , Philips, A. G. , and Hayden, M. R. (1995). Targeted disruption of the Huntington’s disease gene results in embryonic lethality and behavioral and morphological changes in heterozygotes. Cell. 81: 811-823. Cattaneo, E. , Zuccato, C. , and Tartari, M. (2005). Normal huntingtin function: alternative approach to Huntington’s disease. Nat Rev Neurosci. 6: 919-930. Li, J. Y. , Plomann, M. Brundin, P. (2003). Huntington’s disease: a synaptopathy? Trends Mol. Med. 9: 414–420. Reiner, A. , Del Mar, N. , Meade, C. A. , Yang, H. , Dragatsis, I. , Zeitlin, S. , and Goldowitz, D. (2001). Neurons lacking huntingtin differentially colonize brain and survive in chimeric mice. J. Neurosci. 21: 7608-7619. Gauthier, L. R. , Charrin, B. C. , Borrell-Pages, M. , Dompierre, J. P. , Rangone, H. , Cordelieres, F. P. , De Mey, J. , MacDonald, M. E. , Lessmann, V. , Humbert, S. , and Saudou, F. 2004). Huntingtin controls neurotrophic support and survival of neurons by enhancing BDNF vesicular transport along microtubules. Cell. 118: 127-138. Leavitt, B. R. , Guttman, J. A. , Hodgson, J. G. , Kimel, G. H. , Singaraja, R. , Vogl, A. W. , and Hayden, M. R. (2001). Wild-type hungtingtin reduces the cellular toxicity of mutant huntingtin in vivo. Am. J. Hum. Genet. 68: 313-324. Davies, S. W. , Turmaine, M. , Cozens, B. A. , DiFiglia, M. , Sharp, A. H. , Ross, C. A. , Scherzinger, E. , Wanker, E. E. , Mangiarini, L. , and Bates, G. P. (1997). Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation. Cell. 90: 537-548. Arrasate, M. , Mitra, S. , Schweitzer, E. S. , Segal, M. R. , and Finkbeiner, S. (2004). Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death. Nature. 431: 805-810. Frank, S. , and Jankovic, J. (2010). Advances in the pharmacological management of Huntington’s disease. Drugs. 70: 561-571. Bagchi, S. P. (1983). Differential interactions of phencyclidine with tetrabenazine and reserpine affecting intraneuronal dopamine. Biochem Pharmacol. 32: 2851-6 Huntington Study Group. (2006). Tetrabenazine as antichorea therapy in Huntington disease: a randomized controlled trial. Neurology. 66: 366-72 Huggins, M. , Bloch, M. , Kanani, S. , Quarrell, O. W. , Theilman, J. , Hedrick, A. , Dickens, B. , Lynch, A. , and Hayden, M. (1990). Ethical and legal dilemmas arising during predictive testing for adult-onset disease: the experience of Huntington disease. Am. J. Hum. Genet. 47: 4-12. Lea, D. H. , Williams, J. , and Donahue, M. P. (2005). Ethical issues in genetic testing. J. Midwifery Womens Health. 550:234-240. How to cite Mghf, Essay examples